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New Lung "Organoids" in a Dish Mimic Features of Full-Size Lung – Newswise (press release)

By Stem Cell Medical Center

Newswise (press release)
New Lung "Organoids" in a Dish Mimic Features of Full-Size Lung
Newswise (press release)
Newswise New York, NY (May 11, 2017)New lung organoidstiny 3-D structures that mimic features of a full-sized lunghave been created from human pluripotent stem cells by researchers at Columbia University Medical Center (CUMC). The team ...

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New Lung "Organoids" in a Dish Mimic Features of Full-Size Lung - Newswise (press release)

Study Identifies Root Cause of Gray, Balding Hair – NBC 10 Philadelphia

By Stem Cell Medical Center

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Scientists in North Texas have identified the cells that cause hair to turn gray and to go bald findings that could one day help identify possible treatments.

Researchers from the University of Texas Southewestern Medical Center accidentally stumbled upon the discovery while studying a rare genetic disease that causes tumors to grow on nerves.

"When we saw the mice that we were expecting to form a tumor turned gray, we were really excited!" said Dr. Lu Le, an associate professor of dermatology at UT Southwestern.

The researchers found that a protein called KROX20, more commonly associated with nerve development, switches on in skin cells that become the hair shaft.

These hair cells then produce another protein called stem cell factor (SCF).In mice, these two proteins turned out to be important for baldness and graying.

When researchers deleted theSCF gene in mice,the animals' hair turned white; when they deleted the cells that produce KROX20, the mice stopped growing hair and eventually went bald, according to the study.

"We were really excited because as a dermatologist, I treat patients with hair disease, so when we found the root cause of why hair turns gray and hair loss, we just cannot let it go," Le said.

More research is needed to understand if the process works similarly in humans, and Le and his colleagues plan to start studying it in people.

Le hopes that, armed with this knowledge, scientists can develop a topical compound or transplant the necessary gene to hair follicles to correct these cosmetic problems.

Researchers say the findings could one day also provide answers about why humans age in general as hair graying and hair loss are among the first signs of aging.

At Hair Revival Studio in Dallas, clients say the possibility of a treatment for gray hair and baldness sounds remarkable.

"Everyone wants to have a good head of hair. There's a lot of confidence that comes with that," said Brandon Stewart.

Published at 8:55 AM EDT on May 11, 2017 | Updated at 9:54 AM EDT on May 11, 2017

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Study Identifies Root Cause of Gray, Balding Hair - NBC 10 Philadelphia

Uncovering the genetic mechanisms driving embryonic development – Medical Xpress

By Embryonic Stem Cells

May 11, 2017 by Kevin Mccullough Ali Shilatifard, PhD, the Robert Francis Furchgott Professor and chair of the Department of Biochemistry and Molecular Genetics, was the senior author of the study that explored the activation of Hox genes in early embryonic development.

A new Northwestern Medicine study, published in Genes and Development, has identified two DNA elements crucial to the activation of a set of genes that drive the early development of embryos, and which also play an important role in the development of cancer cells.

So-called Hox genes are a related group that control the body plan of a developing embryo; in humans, they regulate the orientation and structure of the vertebrae and spinal cord as well as the location and growth of limbs. Previously, however, the question of how Hox genes become activated, moving from a silent form to an active form, have been poorly-understood by scientists.

"Hox genes are not only crucial for the proper development of the embryo but also play essential roles in tumor formation and metastasis. Understanding the mechanisms that trigger the expression of Hox genes could help us develop novel therapeutic approaches against cancer," said first-author Kaixiang Cao, PhD, a postdoctoral fellow in the laboratory of Ali Shilatifard, PhD, the Robert Francis Furchgott Professor and chair of the Department of Biochemistry and Molecular Genetics.

In the study, the authors present several experiments that provide evidence for a model of embryonic development that utilizes multiple layers of regulation as a "fail-safe mechanism" to guarantee organisms develop properly.

First, the scientists identified two sequences of DNA, located in a so-called "gene desert" between functioning genes, and demonstrated how these sequences ensure activation of Hox genes.

Previously thought to be "junk DNA," the sequences of DNA found in gene deserts have recently been found to play important regulatory roles, and irregularities in these stretches of the genetic code have been associated with disease, including some forms of cancer.

After the scientists pinpointed these previously unidentified DNA sequences, named E1 and E2, they demonstrated they were acting as "shadow enhancers," and regulated the early expression of Hox genes.

Utilizing mouse embryonic stem cell models that had been modified to lack one or both of the sequences, the scientists showed that the two sequences worked redundantly: deletion of either the E1 or E2 sequence resulted in unaffected activation, but removing both E1 and E2 stopped the Hox genes from activating properly.

Separately, the scientists also demonstrated that a protein called SET1A, part of a family of enzymes called COMPASS, which have previously been shown to activate Hox genes, also regulates Hox gene activation: without SET1A, several Hox genes failed to activate.

According to the scientists, the E1/E2 regulation and the SET1A regulation of Hox genes appear to be independent of each other, and are part of a series of multiple regulatory processes that work together to fine-tune the activation of genes essential for the early growth of embryos.

"Future studies that identify small molecules targeting SET1A and factors functioning through the E1/E2 DNA sequences will be important for developing therapies for Hox gene disorders," Shilatifard said.

The project's insight into the process by which Hox genes are regulated, has the potential to identify targets for new treatments for developmental diseases caused by dysfunction in Hox genes, as well as forms of cancer that arise from Hox gene errors, according to the authors.

Explore further: Molecular 'on switch' could point to treatments for pediatric brain tumor

More information: Kaixiang Cao et al. SET1A/COMPASS and shadow enhancers in the regulation of homeotic gene expression, Genes & Development (2017). DOI: 10.1101/gad.294744.116

A landmark study led by UNC School of Medicine researchers has identified the first genetic locus for anorexia nervosa and has revealed that there may also be metabolic underpinnings to this potentially deadly illness.

When a group of researchers in the Undiagnosed Disease Network at Baylor College of Medicine realized they were spending days combing through databases searching for information regarding gene variants, they decided to do ...

A new Northwestern Medicine study, published in Genes and Development, has identified two DNA elements crucial to the activation of a set of genes that drive the early development of embryos, and which also play an important ...

Reported in Nature today, one of the largest sets of high quality human induced pluripotent stem cell lines from healthy individuals has been produced by a consortium involving the Wellcome Trust Sanger Institute. Comprehensively ...

UT Southwestern Medical Center researchers have identified the cells that directly give rise to hair as well as the mechanism that causes hair to turn gray findings that could one day help identify possible treatments ...

Over the last decade, it has made good sense to study the genetic drivers of cancer by sequencing a tiny portion of the human genome called the exome - the 2% of our three billion base pairs that "spell out" the 21,000 genes ...

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Uncovering the genetic mechanisms driving embryonic development - Medical Xpress

Osteoarthritis: Preventing key molecule deficiency may delay need for joint replacements – Medical News Today

By Uncategorized

New research carried out on rodents reveals, for the first time, that preventing the loss of a molecule that is important for cartilage maintenance could delay the onset of osteoarthritis, a disease of the joints that causes disability and affects millions of people in the United States.

Osteoarthritis (OA) - also referred to as degenerative joint disease - is the most common form of arthritis, a general term used to describe a range of conditions that cause pain or disease in the joints.

The new study, led by the NYU Langone Medical Center in New York and published in the journal Nature Communications, shows that injecting the vital cell molecule adenosine into the joints can prevent OA in rat models of the disease.

OA most commonly affects the hips, hands, and knees, and it results from the gradual wear and tear of the cartilage that cushions the ends of the bones in the joint and stops them rubbing against each other.

The degeneration of cartilage in OA progresses slowly and, as time goes by, the joint swells and becomes painful and stiff. In some cases, OA can reduce function and disable a person so much that they cannot lead a normal life.

In fact, the condition can become so disabling - particularly when the knee or hip is affected - that there may be a need for surgery to replace the joint.

The new study concerns various activities of a molecule called adenosine that is essential for cell function and, as the research reveals, is also important for maintaining cartilage.

Cartilage comes from substances secreted by cells called chondrocytes. Having a regular supply of healthy chondrocytes is essential for producing and maintaining cartilage.

The researchers investigated the role that adenosine plays in maintaining a healthy supply of chondrocytes.

Fast facts about OA

Learn more about OA

They note in their study paper that adenosine levels inside and outside cells are "tightly controlled" by cellular stress, oxygen consumption, and the workings of mitochondria - the pockets inside cells that produce the energy for the cell.

Mitochondria supply the cell with energy in the form of chemical units called adenosine triphosphate (ATP), and adenosine is a byproduct of ATP metabolism.

It was already known that aging and inflammation reduces ATP production in chondrocytes, which in turn reduces levels of adenosine. However, until this study, it was not clear how that might relate to OA.

One of the main discoveries of the study is that reduction in chondrocytes, and therefore greater risk for OA, is driven not only by lower levels of adenosine surrounding the cells, but also by loss of signal-receiving proteins called adenosine A2A receptors on the surface of the cells.

If a chondrocyte has no A2A receptors, then it cannot pass the signals from the adenosine molecules surrounding them to their internal machinery. Loss of such signals impairs the cell's ability to maintain cartilage.

The researchers found that mice lacking A2A receptors could not walk as easily as mice with the receptors. On closer examination, they also found OA in the knees of the mice without A2A receptors.

Curiously, the team also found that chondrocytes of rats with OA had raised levels of A2A receptors. They suggest that this is because of a "failed attempt" to make up for the loss of adenosine caused by inflammation-induced changes in ATP metabolism.

When they examined tissue samples of patients who had received replacement joints at NYU Langone because of OA, the researchers found that their chondrocytes also had higher levels of adenosine A2A receptors.

Probing further into the link with ATP metabolism, the team treated mouse chondrocytes with IL-1beta - a molecule that promotes OA. They found that the inflamed chondrocytes produced 39 percent less ATP and showed an 80 percent reduction in a molecule that transports ATP.

The team then showed that reducing adenosine levels by removing the enzyme that allows it to be produced from ATP led to OA in mice. It is known that humans who do not have this enzyme also develop OA.

The researchers also showed that injecting adenosine into the joints of rats with anterior cruciate ligament - a cause of OA in humans - prevented the animals from developing OA.

Senior investigator Bruce Cronstein, a professor of medicine at NYU Langone, says that the study "suggests that diminished ATP and adenosine production are likely contributing factors to the development of osteoarthritis in aging individuals."

He adds that should their findings lead to successful therapies, then they could delay, and perhaps even prevent, the need for the million or so joint replacements that are carried out in the U.S. every year. He also notes that:

"Because joints may have to be replaced again and again, if we can put off the need for joint replacement until later in life, odds are that patients will only have to have this done once."

Learn how knee joint degeneration can be slowed with weight loss.

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Osteoarthritis: Preventing key molecule deficiency may delay need for joint replacements - Medical News Today

Celyad gets FDA Fast Track to Tackle Heart Failure with Cell Therapy – Labiotech.eu (blog)

By Uncategorized

Celyad has received Fast Track Designation from the FDA for C-Cure, a cell therapy aimed to treat heart failure with the patients own stem cells.

Celyad is confident that the announcement of Fast Track Designation from the FDA will help it accelerate the search for a partner to develop C-Cure, a cell therapy for ischemic heart failure. After running a Phase III trial in Europe that didnt meet its primary endpoint, the Belgian biotech identified a subpopulation of more of 60% of patients that did show a significant improvement and is now determined to try again once it finds financial support.

CelyadsC-Cure technology consists in the harvest of the patients bone marrow, which is treated with growth factors and then re-injected into the heart to grow new functional muscle in the area lost due to infarction. The therapy is complementary to Celyads main focus, immuno-oncology. The company is well-known for developing an allogeneic version of the promising CAR-T technologythat recently triggered a license agreement with Novartis.

Image viaJolygon/Shutterstock

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Celyad gets FDA Fast Track to Tackle Heart Failure with Cell Therapy - Labiotech.eu (blog)

This Gun Sprays Stem Cells, Helps Burn Victims Grow Skin in Days – Men’s Health

By Stem Cell Treatment

Men's Health
This Gun Sprays Stem Cells, Helps Burn Victims Grow Skin in Days
Men's Health
A revolutionary new technique is enabling burn victims to heal quicker, less painfully, and with more normal skin. And it's all thanks to a gun. The SkinGun sprays stem cells onto wounds and allows patients to grow a new, healthy layer of skin in as ...

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This Gun Sprays Stem Cells, Helps Burn Victims Grow Skin in Days - Men's Health

Stop MS: Buena Vista’s Chris Martin set to try cutting-edge stem cell program – Chaffee County Times

By Stem Cell Treatment

Buena Vista resident Chris Martin just wants to stop the progression. Diagnosed with multiple sclerosis almost 11 years ago, Martin is set to begin his first round of experimental umbilical stem cell therapy from Denver Regenerative Medicine next month.

Martin experienced his first symptoms in 2006, while running the Super 8 motel in town. He was having difficulty counting money, using his hands and walking.

Initially Martin and doctors thought it could have been a stroke or a tumor, but that was quickly shown not to be the case following an MRI.

While the initial MRI showed some signs of potential multiple sclerosis, doctors cant diagnose patients with the disease until additional symptoms or followup attacks are shown. It took another 2 years before Martin was diagnosed.

Whats terribly frustrating with all these diseases, whether its Parkinsons, MS or Alzheimers, is they dont really know what causes it, why it goes fast, they dont know why it goes slow (in some cases). They dont know if Copaxone is going to be good for you or if Rebif is going to be good for you. They just dont know. It was just a big shrug of the shoulders, Martin said about initially dancing through the minefield of doctors and diagnoses when he was first seeking treatment.

Rebif and Copaxone are both medications used to treat MS, both prescribed to Martin at various times over the years.

In laymens terms, MS happens when your immune systems attacks the fatty myelin materials that wrap around your protective nerve fibers, making it difficult for you brain and body to communicate effectively.

Since the nervous system doesnt work as intended, sufferers of MS can experience muscle spasms, numbness and tingling in parts of the body and trouble walking, among many other symptoms.

It was probably about 2015 that my leg started twitching at night and not really a lot of pain, just discomfort especially in my left leg which feels like it weighs about 400 pounds, Martin said, while manually stretching out the leg with his right arm. At least in this stage of MS, its not terribly painful, but I started to get symptoms of twitching at night time which is pretty common. The only thing that really seemed to calm it down was medical marijuana.

To help sleep, Martin usually takes an indica strain marijuana edible at night to help with the twitching and to get restful sleep, he said.

Indica strains, as opposed to the sativa strains, are know for their physically sedating effects and can help with physical and mental relaxation.

Martin also found some solace in juicing the leaves of marijuana plants, but said it became too difficult because of the amount of leaves needed for the juice.

Doctors do not yet know what causes the disease, but research into various forms of stem cell therapy have shown promise to ease symptoms and to slow or even stop the progression of the disease.

Martin began looking into clinics and stem cell therapy options last year.

Various forms of stem cell treatment exist, including bone marrow transplants and a form of stem cell therapy that utilizes cells taken from adipose tissue, or fat cells, harvested from the patients own body. After talking with several clinics and doctors, Martin decided on umbilical stem cell therapy.

The treatments, however, are costly and Martins insurance provider will not cover the experimental procedure.

None of these stem cell clinics claim to cure anything for obvious reasons. Some people maybe dont get anything from it (treatment), maybe they get a little bit better. So if you get a little bit better and spend $20,000, is the patient happy or not happy? Its a quality of life question, Martin said.

Martin met with a doctor at Denver Regenerative Medicine who explained the process.

A bio company harvests the stem cells and processes them before shipping them to doctors practicing regenerative medicine across the country. Then the stem cells are injected through an IV.

He will be only the third MS patient to receive this form of stem cell therapy from the doctor administering it, Martin said.

The company harvesting the stem cells is Burst Biologics, Martin said.

One patient that had received stem cell treatment from Denver Regenerative Medicine told Martin that the first 24 hours after the stem cell injection, she had a euphoric experience.

She was walking well and had even cleaned her entire house after relying on a wheelchair and cain prior to the injection. After the initial 24 hours, however, she told Martin that the effects from the injection had begun to go away and she felt like she had prior to the injection.

After about a month, she started to feel good again and shes no longer in a wheelchair and shes walking really well with her cane, Martin said she told him. She highly recommends it. She just cant believe how good shes feeling.

Another patient from Denver Regenerative Medicine told Martin that after his injection and a followup injection, he hasnt seen much of an effect.

My gut feeling is the doctors good because hes kind of on the leading edge, but hes not the key to this stem cell. Its the biology company, in my opinion, is the brains behind all of this because theyre the ones harvesting the stem cells, processing it and getting it ready, Martin said.

With around a month before Martin begins his first treatment, he said hes solely focused on getting his body in the best shape possible.

I want to get my body as strong as possible to react to the stem cells the best that it can. And that involves more trips to the gym, getting my hormones balanced correctly and making sure that I dont miss a dose of any of my nutritional medicines, Martin said.

A little over a decade since Martin was diagnosed, he said he can feel the progression of the disease gradually increasing day-to-day.

By trade, Martin is an engineer, making the mysterious nature and complications of the disease difficult to come to terms with since there is no workable or obvious solution, but maybe the stem cell injection will help. Hes optimistic.

Its gotten worse in the last 2 years, so my ability to move in the last 2 years has been hard. Now its been almost 11 years and the last two years, its definitely slid faster than the previous 9. As an engineer, I think about it as accelerating and I ask the doctor why and its I dont know, Martin said. Statistically, the doctor did say in the old days, you either go down relatively quick, like a 25-year-old gets MS and theyre in a wheelchair at 27 or they do pretty well for 10 years and then it starts to drop off. And Im at my 10 years, so yeah, Im concerned about it dropping off. I would like it to stabilize and I would like to get the word out to other people from Parkinsons to MS to neurological mysteries to keep pushing the industry forward. Im excited about it, thats the bottom line.

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Stop MS: Buena Vista's Chris Martin set to try cutting-edge stem cell program - Chaffee County Times

Vail Daily column: School district has model partnership with medical center – Vail Daily News

By Stem Cell Medical Center

Our school district goes out of its way to develop meaningful and impactful partnerships with all kinds of groups in our valley. We understand that we can't solve all of our community's problems on our own. That's why we've reached out to community organizations to try to develop systems of support for all of our students.

One model partnership we are very proud of is the collaboration and support we get from Vail Valley Medical Center. On top of providing quality medical care, VVMC is also very good to our kids and schools.

One major area where VVMC supports our students is through medical coverage for our athletes. Annually, VVMC provides $188,000 in support and medical care, including two certified athletic trainers who are available to our athletes.

VVMC also supports a program called ThinkFirst, which teaches students about preventable injuries. Sure, kids will be kids, but ThinkFirst is a proven program designed to help students think through the consequences of their actions before leaping.

VVMC also hosts field trips from schools to the hospital and research labs on a regular basis. Students get to see the medical profession in action, as well as see some of the cutting-edge research VVMC is doing around sports medicine and stem-cell work. These tours give our students an understanding of what a career in medicine might look like and what kinds of jobs might be available for their futures.

VVMC sponsors a program aimed at reducing skin cancer and premature aging through its Sun Safety program. This program introduces elementary school children to the dangers of overexposure to sun and provides sunscreen free to the students and their families.

VVMC is also a big supporter of our community education foundation, the Education Foundation of Eagle County. By supporting the foundation and events such as Project Funway and Wild West Day, VVMC provides classroom and instructional supports that go directly to students.

Looking ahead, we're excited about working with VVMC to also add career/technical pathways for our students into the medical profession. Through experience-based learning opportunities such as apprenticeships and internships, as well as high school credit courses in the medical field, our partnership with VVMC can help us provide a great on-ramp to a career in the medical field.

It really does "take a village" to raise a child, and we're proud of our partnership with VVMC as a model example of how the community school system and the community health care system can work together for the betterment of kids in Eagle County.

Jason E. Glass is the superintendent of Eagle County Schools. He can be reached at jason.glass@eagleschools.net.

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Vail Daily column: School district has model partnership with medical center - Vail Daily News

Stem Cell Vaccine Found to Increase Immune Responses, Inhibit Tumors – University of Cincinnati

By Stem Cell Medical Center

Stem Cell Vaccine Found to Increase Immune Responses, Inhibit Tumors

CINCINNATIResearchers at the University of Cincinnati (UC) have found that a cancer stem cell vaccine, engineered to express a pro-inflammatory protein called interleukin-15 (IL-15) and its receptor (IL-15Ralpha), caused T cell production in animal models and enhanced immune responses against tumors.

This T cell production showed a cellular immune response that could lead to new immunotherapy treatments for cancer with improved side effects.

These findings are being presented via poster abstract at the American Society of Gene and Cell Therapys annual meeting in Washington, D.C., May 10-13.

"IL-15 is a powerful stimulator of the maturation and activation of T cells and natural killer cells that recognize and attack tumor cells. Human IL-15 was first used in Phase I clinical trials to test its efficacy for treatment of a number of cancers, including melanoma and kidney cancer, but caused a number of side effects that made high doses difficult for patients to tolerate, says John Morris, MD, co-author of this study, clinical co-leader of the Molecular Therapeutics and Diagnosis Program for the Cincinnati Cancer Consortium, co-leader of the UC Cancer Institute's Comprehensive Lung Cancer Program, professor in the Division of Hematology Oncology at the UC College of Medicine and UC Health medical oncologist.

"In this work, we showed that transferring the genes for both IL-15 with its receptor into cancer cells increased the cell-surface presentation of IL-15 to T cells, and in turn, stopped the tumor cells from reproducing with little evidence of side effects in animal models.

"In an effort to enhance antitumor activity and reduce side effects, we studied a vaccine targeting cancer stem cells, the cells in a tumor thought to be resistant that give rise to recurrent tumors after treatment, by genetically altering them to express IL-15 and IL-15Ralpha to see if lung cancers implanted in animal models shrunk.

Using animal models and their lung cancer cell lines, researchers introduced the IL-15/IL-15Ralpha-modified lung cancer stem cells as a vaccine and saw dramatically inhibited tumor growth.

"Animal lung cancer stem cells expressing IL-15 and IL-15Ralpha stimulated proliferation of T cells suggesting the ability to enhance immune responses, he says. "These findings further support evidence of IL-15s ability as a cancer treatment. We are continuing vaccination studies in animal models with hopes of moving this research to a Phase I trial in humans to see if side effects are reduced.

Donatien Toukam, PhD, post-doctoral fellow in the Division of Hematology Oncology, is the lead author of this study funded in part by the Lcs Foundation. He cites no conflicts of interest.

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Stem Cell Vaccine Found to Increase Immune Responses, Inhibit Tumors - University of Cincinnati

Tucson Health and wellness events, support groups and more – Arizona Daily Star

By Stem Cell Medical Center

HEALTH AND WELLNESS

All events are free unless otherwise noted.

Widowed to Widowed The Fountains at La Cholla, 2001 W. Rudasill Road. Offers group discussion and support. 10-11 a.m. Mondays. 797-2001.

Interfaith Spiritual Support Group Parlor at First Christian Church, 740 E. Speedway. An interfaith spiritual support group for adults affected by mental health disorders. 6-7:30 p.m. May 15. 624-8695.

Healthy Living with Chronic Pain Desert Harmony Hospice of Tucson, 1200 N. El Dorado Place. Six-week interactive workshop for individuals living with chronic pain and their caregivers. Ages 18 and up. 1-3:30 p.m. Tuesdays. $35. 305-3410.

Taekwondo Wellness Intuition Wellness Center, 5675 N. Oracle Road. Learn traditional Taekwondo, philosophy and core principles, self-care, stress management, coping skills, social skills and mindfulness meditation. 4:15 p.m class for ages 7-12; 5:15 p.m. for ages 12 and up. 4:15-5:15 p.m. Tuesdays and Thursdays; 5:15-6:15 p.m. Tuesdays and Thursdays. $20. 333-3320.

Right treatment, right person, right time: Progress in precision cardiovascular care at the Sarver Heart Center Banner-University Medical Center Tucson, 1501 N. Campbell Ave. Learn about human "disease in a dish," induced pluripotent stem cell (iPSC) technology that has the potential to completely change medicine in terms of understanding individual variation, underlying disease mechanisms, transforming and personalizing drug discovery and targeting treatments, sometimes at the level of correcting a single gene mutation. 6-7:15 p.m. May 17. 626-2901.

Beyond Movement: Cognition and Emotion PWR! Gym, 140 W. Fort Lowell Road. Marisa Menchola will introduce the most common cognitive and emotional changes experienced by people with Parkinson Disease. 4-5:30 p.m. May 18. 591-5346.

Experience Wellness: A day of activities and education Thrive Wellness Collective, 4737 N. First Ave. Free mini treatments, demos, lectures, snacks and door prizes. 11 a.m.-3 p.m. May 20. 477-2107.

Free Eye Exams St. Elizabeth's Health Center, 140 W. Speedway. Will screen for cataracts, glaucoma, macular degeneration, dry eyes and complications related to diabetes. Call to make an appointment. 8 a.m.-noon. May 20. 694-1471.

Depression: Share your story to help someone else get through it... Call for guests on The Depression Session The Depression Session on Downtown Radio, One E. Toole Ave. Share your story of Depression on 99.1FM Downtown Radio, and help de-stigmatize depression. Prerecorded and edited. Contact: lmilkins@gmail.com. Ages 18 and up. Noon-12:30 p.m. Sundays.

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Tucson Health and wellness events, support groups and more - Arizona Daily Star

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